By Amy L. Sutton
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Extra resources for Alzheimer Disease Sourcebook, 5th Edition
Some families with CJD, GSS, or fatal familial insomnia have mutations in the prion protein gene, although these disorders can also occur in people without the gene mutation. Individuals with these mutations are at signiﬁcantly higher risk of developing these forms of dementia. Abnormal genes are also clearly implicated as risk factors in Huntington disease, FTDP-17, and several other kinds of dementia. Smoking and alcohol use: Several recent studies have found that smoking signiﬁcantly increases the risk of mental decline and dementia.
They do know that plaques and tangles usually increase in the brain as AD progresses. In the early stages of AD, patients may experience memory impairment, lapses of judgment, and subtle changes in personality. As the disorder progresses, memory and language problems worsen and patients begin to have difﬁculty performing activities of daily living, such as balancing a checkbook or remembering to take medications. They also may have visuospatial problems, such as difﬁculty navigating an unfamiliar route.
Metabolism All cells break down chemicals and nutrients to generate energy and form building blocks that make new cellular molecules such as proteins. This process is called metabolism. To maintain metabolism, the brain needs plenty of blood constantly circulating through its billions of capillaries to supply neurons and other brain cells with oxygen and glucose. Without oxygen and glucose, neurons will quickly die. Repair Nerve cells are formed during fetal life and for a short time after birth.
Alzheimer Disease Sourcebook, 5th Edition by Amy L. Sutton